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To directly assess insulin signaling, mice were administered insulin via the vena cava, followed by immunoblotting of key downstream effectors in liver and eWAT

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Regarding inflammatory signaling, GHK-Cu peptide appeared to mitigate the NF-B pathway by suppressing phosphorylation of the p65 subunit at Ser536 and blocking its nuclear translocation

6-AH is a metabolically stable antagonist to the HGF/c-Met system that works by preventing HGF dimerization and activation (Kawas et al., Statistical analysis Data were analyzed using GraphPad Prism (V